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In addition to lipid uptake, tumorigenesis induced chronic stress activates the -adrenergic receptor (2-AR) in MDSCs to enhance FAO and upregulate the mitochondrial fatty acid transporter CPT1A, thereby reinforcing their PGE2 mediated immunosuppressive capacity (77, 78)

(1) Systemic entry and uptake: Tyramine escapes normal intestinal and hepatic metabolism, enters the circulation, and is transported into sympathetic nerve terminals via the norepinephrine transporter (NET)

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