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The GCH1/BH4 pathway acts as an endogenous antioxidant pathway, GCH1 protects cells from ferroptosis primarily through the antioxidant action of BH4, and is completely independent of GPX4-mediated protection against ferroptosis [44]

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Notably, the mechanism of human GSTO1-1 may be that the backbone amide group of Phe34 donates a hydrogen bond to the sulphur atom of the H-site bound GSH 18

Equivalent results were also obtained using hypoxanthine phosphoribosyltransferase (HPRT)

Elkholy, N., Hariri, M

Insulin syringes are marked in units, where 100 units equal 1 mL