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In this way, it provides a controlled model for studying fat metabolism and energy regulation

They are a clinically established treatment for B12 deficiency

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The peptide also does not stimulate IGF-1 production, avoiding the growth-promoting and potentially mitogenic effects associated with IGF-1 elevation

To clarify their relative importance, we examined Cyp7a1 induction in response to oral glucose loading in Kir6.2 deficient mice ( Kir6.2 / mice), which lack glucose-stimulated insulin secretion 13

Treatment with methylcobalamin or hydroxocobalamin may unmask this condition