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This suggests that CaOx-exo exacerbates pre-existing renal injury caused by kidney stones, potentially through enhanced macrophage phagocytosis or upregulated expression of scavenger receptors, thereby increasing the uptake of exogenous exosomes [32]

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UPR-driven tumor dormancy and therapeutic resistance Beyond its role in active tumor growth, the UPR plays a pivotal role in cancer cell dormancy, long-term survival, and resistance to anticancer therapies

The placenta manages a complex exchange of nutrients and antioxidants to ensure the baby has what it needs

KEAP1 (kelch like ECH associated protein 1), an enzyme often mutated in lung cancer, interacts with NFE2L2, leading to NFE2L2 degradation [154, 155]

Getting to know ovarian cancer ascites: opportunities for targeted therapy-based translational research