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Recent studies have shown that autistic disorder is likely to involve multiple genes [79] although a common genetic change is not seen in all cases suggesting that it is likely to be a cluster of conditions, each with its own individual and yet overlapping pathology

However, the exact mechanism remains to be illustrated

OxLDL components and their interaction with toll-like receptors (TLRs) 2 and 4, CD36 and other cellular receptors further mediate thromboinflammation enhancing tissue and organ damage culminating in organ failure, i.e., myocardial infarction, stroke, and pulmonary artery embolism (174, 209212)

In the present study we investigated the role of glutathione in the response of A