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This process is closely associated with ROS production induced by iron overload and p53 acetylation (Stockwell et al., 2017)

However, a similar analysis for the change in platelet count from beginning of phase 1 to the beginning of phase 2 was performed, with excretion of toxic metals after the 9 th dose of DMSA and change in glutathione as the dependent variables

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Low levels of reactive oxygen species (ROS) mediate signaling cascades involved in cell proliferation [8], while high ROS levels can lead to DNA damage and eventual cell death [9]

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