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therefore, bacterial GGTs have been largely expressed heterologously in E

One of which is the stability and bioavailability of orally administered NAD + precursors, which induce slow and suboptimal NAD + increases 39,69

5d) therefore suggests a less efficient interaction between E170X mutants and HEDS-derived GSSEtOH in accordance with the scaffold model and the hypothesis that GSSEtOH formation is catalysed by ScGrx7

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