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The primary mechanisms we see cited in the literature include: VEGF upregulation: BPC-157 increases vascular endothelial growth factor, which drives new capillary formation in injured tissue

Although there are 8 isoforms of GPx, GPx1 and GPx4 seem to be the most studied

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Our results showed that the deposition of aldehyde adducts is present within the earliest histological lesions of the hepatocellular injury in the Aldh2*2 KI mice after AA exposure, suggesting the involvement of the aldehyde storm in the exacerbation of acute hepatocellular injury