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This combined effect of metformin on glycerol-3-phosphate dehydrogenase, substrate inhibition, and GLP-1 secretion makes it an essential therapeutic option for controlling hepatic gluconeogenesis and maintaining glucose homeostasis in individuals with insulin resistance or T2DM (Bahne et al., 2018)

Ruenzi M, Stolte M, Veljaca M, Oreskovic K, Peterson J

This mechanism supports studies in cellular rejuvenation, energy production, and metabolic resilience

And when those things are out of balance, you can start to notice increased levels of inflammation in your body

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