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Nevertheless, experimental data suggest that the altered intracellular redox status associated with hyperhomo-cysteinemia-induced oxidant stress is the major molecular mechanism responsible for the atherothrombogenicity of moderately elevated circulating homocysteine

Serum HO-1 levels of patients newly diagnosed with acute exacerbation of ILD were measured at the time of initiating steroid pulse therapy

Sabdyusheva Litschauer, I

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Preclinical animal studies suggest it may promote mucosal healing, reduce inflammation, and support intestinal barrier repair

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