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Therefore, a "link model" approach was used to implement the PK/PD model and to estimate the drug concentration in the effect-site compartment [24]

In essence, MOTS-c sends a signal to cells to increase energy expenditure and reduce excess fat storage

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Future research should prioritize mechanistic studies using knockout and CRISPR-based models to precisely define the cellular pathways through which GLP-1RAs exert their anti-inflammatory and metabolic effects in the lung