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Figure 1 Table 1 Correlations of GSTM3 expression in tissues and clinicopathological parameters

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Current therapies include limiting absorption of APAP by administering activated charcoal 8,9,10 , replenishing glutathione stores by administering the glutathione precursor n -acetylcysteine (NAC) 11,12 , the JNK/CYP2E1 inhibitor fomepizole ( 13 ), and supportive care

For example, SFN is one of the most potent naturally occurring inducers of phase II enzymes, contributing to chemoprevention (115)

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This leads to meiotic defects, particularly in oocytes from aged females, highlighting the age-dependent susceptibility to oxidative damage [84]