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Relative to the wild-type enzyme, mutation of residue 15 to serine or alanine in UGT74F2 did not lessen the specific activity for SGE formation, but did increase the formation of SAG

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In contrary to the brain, kidneys, and small intestine, other organs are not able to take up intact GSH due to a lack of the Na + -dependent GSH transporter at luminal endothelial cell membranes (Fig