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Women are frequently diagnosed and uniquely at risk because pregnancy heavily stresses the bodys energy demands and further depletes carnitine levels, leading to a metabolic crisis [1] [8]

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Still in the development pipeline are nonlethal small-molecule inhibitors, or drugs that block the second step in the pathway, the major host gene responsible for converting microbe-generated TMA into TMAO, flavin monooxygenase 3

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doi: 10.1371/journal.ppat.1000758 229 LevitzS