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These secreted factors recruit circulating monocytes to the site of endothelial injury and induce the transformation of monocytes into pro-inflammatory macrophages, which in turn develop into foam cells, promoting the formation of atherosclerotic plaques at the site of injury (66)

Increased permeability supports nanoparticle extravasation in selected regions, while poor perfusion, high interstitial pressure and heterogeneous flow limit uniform delivery across the tumor mass [17, 33]

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Obesity and polycystic ovary syndrome: implications for pathogenesis and novel management strategies

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Kunimatsu J, Suzuki TW, Ohmae S, Tanaka M