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AAT has inhibitory activity against all NSPs but is particularly active against NE (57)

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2.3Nonalcoholic fatty liver disease and NRF-2 To define nonalcoholic fatty liver disease, it should be proven, by means of an image or a histological method, that the steatosis is not related to any secondary cause that conditioned the accumulation of liver fat, such as alcohol consumption, infection by hepatitis C virus, or any congenital metabolic disorder, steatogenic medications or among other causes

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