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The METH-induced brain injury significantly increased lipid peroxidation and decreased the level of the thiol group, the glutathione total and the activity of brain antioxidant enzymes (catalase, superoxide dismutase and glutathione peroxidase)

doi: 10.1515/hsz-2016-0202 90 FraternaleA.PaolettiM

Fate mapping analysis of lymphoid cells expressing the NKp46 cell surface receptor

The principal function of endogenous GSH is not to limit inflammation but to fine-tune the innate immune response to infection (Diotallevi et al., 2017

The Journal of Nutrition, 134 (3), 489-492

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