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To test this hypothesis, we constructed the gpoA Cys36Ala mutant, which demonstrated a higher susceptibility to H 2 O 2 in reference to the wt strain, a similar phenotype displayed by the gpoA mutant (Fig 3B, Fig S2)

Furthermore, animal models have empirically proven that maintaining a high ratio of charcoal to toxin (ideally a 10:1 ratio) is necessary to push the chemical equilibrium toward the bound complex, ensuring that the toxins do not detach from the charcoal further down the digestive tract

[13] Orally ingested compounds first pass through the gastrointestinal tract and gut microbiome where some xenobiotics can be metabolized before entering the bloodstream, while inhaled, intravenous, or dermal exposures enter circulation more directly

Iranian Journal of Basic Medical Sciences, 20 (5), 557-568