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from the Intramural Research Program of the National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases (ZIA DK075062)
54 Via a peripheral mechanism, this is thought to contribute to lower insulin sensitivity and the development of insulin resistance
Collectively, these antioxidants contribute to the overall defense against oxidative stress in the gut, preventing cellular damage and inflammation
For example, insulin can promote the synthesis of phosphatidylcholine through the upregulation of the Kennedy pathway, while glucagon can induce the degradation of phospholipids as part of the body's response to fasting
Secondary mechanisms growth factor modulation (EGF, FGF) and nitric oxide system interaction have a smaller evidence base and are considered less central to BPC-157's research profile
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