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Lang CH, Nystrom GJ, Frost RA

Instead of cycling through flare and recovery, the joint stabilizes at a lower baseline of inflammation

[44] [45] Although no specific PA susceptibility genes have been identified, a genetic factor likely is involved in the disease

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The tendon-to-bone healing interface is notoriously weak, and retear rates remain high even after technically successful surgery

For example, rats treated with the benzodiazepine antagonist flumazenil along with a benzodiazepine do not develop tolerance but still apparently experience an anxiolytic effect