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SS-31 stabilizes mitochondrial structure and function, prevents the collapse of mitochondrial membrane potential, and inhibits calcium-induced opening of the mitochondrial permeability transition (MPT) pore

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Experimental vascular studies using isolated rat aorta preparations reported that BPC-157 modulated vasomotor tone and increased nitric oxide generation through activation of the SrcCaveolin-1endothelial nitric oxide synthase (eNOS) signaling pathway [3]

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