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For the systemic injury and recovery uses that TB-500 is actually marketed for, the supporting data are animal models and in-vitro work on thymosin beta-4 and its actin-binding domain

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Uzasadnienie czenia obu peptydw w protokoach badawczych opiera si na ich komplementarnych mechanizmach molekularnych: TB-500 oddziauje na cytoszkielet aktynowy i migracj komrkow, podczas gdy BPC-157 moduluje angiogenez przez szlak NO/VEGF