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Neuropathology of AD involves the accumulation of senile plaques (protein fragments of amyloid-, A) and neurofibrillary tangles (NTFs, deposits of phosphorylated protein tau) in the brain

Endothelial/angiogenic signaling involving VEGFR2AKTeNOS, nitric-oxidelinked vasodilation, and cell-migration pathways (FAKpaxillin) in tendon fibroblasts

The performance is influenced by the worm burden indicated by the infection intensity through counting eggs in feces, with low accuracy in light-intensity infection (250, 251)

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As the cellular antioxidant capacity weakens and lipid ROS accumulate, the redox balance within cells is thereby disrupted, which induces cell death (20)

Many different methodologies have been used