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This multidimensional network of interactions positions AMPK as a central hub linking energy sensing, metabolic regulation, and inflammatory responses, offering multiple therapeutic targets for the precise intervention of NAFLD

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[9] The Long R3 design pushes the same principle further: the Arg-for-Glu swap at position 3 removes a residue important for binding-protein contact, and the 13-residue N-terminal extension further perturbs the IGFBP interface