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Sido et al, 1998), the increase in -GCS in mucosa from TNBS-treated rats can be interpreted as an adaptive response to control the consequences of the stress induced by TNBS as -GCS-HS is known to be upregulated in response to divergent stressful conditions including oxidative stress or GSH depletion (Morales et al, 1997, 1998

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Since some of the main components of ME/CFS include redox imbalance, chronic inflammation, and defective energy metabolism, it is also important to investigate how these components interact bidirectionally (201)

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57 The accumulation of DCs in the articular cavity has also been reported