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According to the metabolic spectrum, the main bile acid of HT patients is DCA, which prompts the possible relationship between DCA and the overgrowth of intestinal bacteria in HT patients [196]

It is instrumental in modulating the inwardly rectifying K+ current and action potential duration in cardiac muscles [11], along with inhibiting the fast Na+ current, thereby evoking class I antiarrhythmic activity [12]

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