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In the pre-synaptic neurons, the GABA-T converts the GABA into succinic semialdehyde (SSA) and, therefore, makes the neuron GABA deficient and elevates the glutamate levels, which are excitatory neurotransmitters (Lu et al., 2023)

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1 Introduction Periodontitis is a globally prevalent chronic inflammatory disease triggered by dysregulation of oral biofilm and host immune responses, serving as the primary cause of adult tooth loss ( A multi-platform analysis of human gingival crevicular fluid reveals that ferroptosis is an associated regulated cell death mechanism during the clinical progression of periodontitis ( Porphyromonas gingivalis can actively induce ferroptosis through multiple pathways ( This review systematically summarizes the molecular mechanisms of ferroptosis in periodontitis, with a focus on how dysregulated iron metabolism, reprogrammed lipid metabolism, and suppressed antioxidant defenses in the periodontal microenvironment regulate ferroptosis

Rigorous research has been conducted to unravel the complexities of GPS, employing advanced methodologies such as high-performance liquid chromatography (HPLC) for chemical profiling and cell-based assays for evaluating biological activities

Torres AR

PMID: 27687962