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Description

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Glucagon is traditionally thought of as the opposite of insulin

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The observation that BPC-157 appears to activate this pathway (via phosphorylation of FAK and paxillin) in cultured fibroblasts provides a direct molecular explanation for how it might accelerate wound closure and tissue repair in research models

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Cheetham TD, Holly JMP, Baxter RC, Meadows K, Jones J, Taylor AM, Dunger DB

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