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Although these compounds were originally developed to block histone acetylation, their ability to dampen CBP/p300-dependent acylation marks at promoters and enhancers raises the possibility that catalytic CBP/p300 inhibitors might also act as indirect anti-lactylation agents, especially in settings where p300/CBP contribute substantially to histone lactylation

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doi: 10.1167/iovs.02-0822