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If endometriosis is partly a metabolic disease, then metabolic medications are not treating a side effect

Cancer 97(5):1234-41, 2003

This unexpected closed form without a substrate or product could result from the binding of the buffer MES molecule in the active site

highlighted the potential of PROTACs to overcome drug resistance and target undruggable proteins, citing ARV-110 and ARV-766 (another AR-directed PROTAC in phase II trials for CRPC) as exemplars (Malarvannan et al., 2025)

Dias IHK, Marshall L, Lambert PA, Chapple ILC, Matthews JB et al