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ROS function as dual agents in the cellular environment

First, under acidic conditions, NO 2 can be protonated to form nitrous acid (HNO 2 ), which spontaneously decomposes to generate nitric oxide (NO)

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Filling the tank: keeping antitumor T cells metabolically fit for the long haul

This dual dysfunction reduces glucose uptake and antioxidant defense while paradoxically activating mTORC1 to promote cellular senescence (32, 33)

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