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Akt is deactivated and Bcl-2-associated death promoter (BAD) phosphorylation is lost in one pathway due to a stalled survival pathway, whereas Fas cell surface death receptor (FAS) is upregulated through stimulation of c-Jun-N-terminal kinase, which activates caspase-8, phosphorylates Fas-associated protein with death domain (FADD), and truncates BH3-interacting domain death agonist (BID)
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Review: the AngIV-analog development programme for Alzheimer's and Parkinson's Development programme review A review describing the programme to develop angiotensin IV analogues as agents against Alzheimer's and Parkinson's disease
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