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Inflammation in the CNS of patients with MS or EAE is known to be primarily mediated by T helper type 1 (Th1) (67, 68) and Th17 T-cell (6971) subsets that produce proinflammatory cytokines, such as TNF- and IL-17, which have suppressive effects on the T regulatory (Treg) anti-inflammatory T-cell subset

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doi: 10.1172/JCI88881 199 SchwabeRFTabasIPajvaniUB

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