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Mechanism of action is mitophagy activation (selective recycling of dysfunctional mitochondria), with preclinical data showing ~45% lifespan extension in C

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The lesions may or may not parallel luminal activity

Some researchers cycle thymosin alpha-1, while others maintain continuous low-dose protocols

It also regulates matrix metalloproteinases (MMPs), particularly MMP-2 and MMP-9, which are involved in extracellular matrix remodeling during tissue repair