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In spite of their limitations, these studies are the first to demonstrate a capacity of exogenously administered NAC to reach the CNS

Specifically, the addition of cuprizone (bis-cyclohexanone oxalydihydrazone), to the diet process (66) produces a massive demyelination in different areas of the CNS and particularly in the corpus callosum (67-69)

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Furthermore, reactivity is strongly enhanced if neighboring residues are basic (positively charged), whereas acidic residues in the vicinity of the sulfhydryl group oppose glutathionylation

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