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In transected rat Achilles tendon models, BPC-157 treatment resulted in accelerated recovery with increased load to failure, superior functional scores, enhanced cellular infiltration for repair, better collagen fiber organization, and smaller persistent defects compared to untreated controls

Available at: Summary Keywords dipeptidyl peptidase 1, cathepsin C, elastase, neutrophil, inflammation, pathophysiology, treatment strategy, respiratory disease Citation Chalmers JD, Kettritz R and Korkmaz B (2023) Dipeptidyl peptidase 1 inhibition as a potential therapeutic approach in neutrophil-mediated inflammatory disease

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Forma: Stabilny, mikronizowany izolat

Interest in such applications is largely driven by their proposed mechanisms of action, including enhancement of tissue repair, modulation of inflammation, and cytoprotective effects observed in preclinical studies

(2) Maar K, Hetenyi R, Maar S, Faskerti G, et al