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Structural changes in cerebral microvasculature induced by ferroptosis contribute to blood-brain barrier disruption in Alzheimers disease: an autopsy study

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This results from a tyramine-induced depletion of norepinephrine normally present in the readily releasable vesicle pool, and replacement of vesicular norepinephrine with tyramine (which functions as a false- or pseudo- neurotransmitter), which when released has no effect on postsynaptic adrenergic receptors (Figure 1)

10.1371/journal.pone.0034263 149 SkopelitouK.MuletaA

Journal of Pharmacology and Experimental Therapeutics 303 , 10611066 (2002)