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Even modest increases in hematoma formation, persistent drainage, or delayed wound healing may amplify susceptibility to PJI, a complication with disproportionate morbidity and resource utilization
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Mechanistically, BPC-157 appears to function through nitric oxide-mediated vasodilation, angiogenic factor regulation, and recruitment of fibroblasts and endothelial progenitor cells 1-3
Peptides support healing but do not replace necessary structural interventions
Direct supplementation with Urolithin A overcomes limitations of dietary exposure and gut microbiome variability in healthy adults to achieve consistent levels across the population
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