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It tends to upregulate genes involved in regeneration and downregulate those associated with inflammation or fibrosis

Untreated pernicious anemia can cause vitamin B12 deficiency leading to megaloblastic anemia and neurological disorders, even in the presence of adequate dietary intake of vitamin B12. 7

[115] This functionality is lost in vitamin B 12 deficiency, resulting in an increased homocysteine level and the trapping of folate as 5-methyl-tetrahydrofolate, from which THF (the active form of folate) cannot be recovered

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They are not interchangeable, and the comparison is not symmetrical