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European Journal of Clinical Pharmacology, 55(2), 111-115

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The homocysteine-inducible, endoplasmic reticulum (ER) stress-inducible, ubiquitin-like domain member 2 (HERPUD1) in cultured HELA cells facilitate cytoprotective effects against oxidative stress by suppressing the inositol 1,4,5-triphosphate receptor and reducing the transfer of calcium ions from the endoplasmic reticulum to mitochondria [41]

Abstract Background/aims Serious adverse event reporting guidelines have largely been developed for pharmaceutical trials

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The interaction between oral and gut microbiota can influence the brain through the gut-brain axis, including disturbances in neurotransmitter metabolism, activation of systemic immune responses, and direct or indirect effects of bacterial metabolites (such as short-chain fatty acids, lipopolysaccharides, etc.) on the bloodbrain barrier and neural function