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These polymorphisms are unrelated to cobalamin metabolism and the elevated MMA levels do not, by themselves, indicate a B12 deficiency (Molloy, 2016)
Unlike the molecular design of brensocatib and BI 1291583, HSK31858 employs a non-peptide non-covalent inhibition mechanism, binding to DPP1 through non-covalent interactions with an IC50 of 57.4 nM ( Structural biology research reveals the unique binding mode of HSK31858 with DPP1
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Release of glycerol, however, would be impaired
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