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This review integrates recent advances in our understanding of the distinct yet interconnected pathological processes affecting articular cartilage, subchondral bone, synovium, infrapatellar fat pad, menisci, ligaments, and peri-articular musculature

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Studies have shown that regulating oxidative stress and antioxidant enzyme expression, such as enhancing manganese superoxide dismutase (MnSOD) and GPx activity, can effectively slow the progression of muscle atrophy, providing a new approach to sarcopenia prevention (73)

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As mitochondrial efficiency declines and ROS accumulate with age and disease progression, cells experience increased molecular damage and impaired neurophysiological function, including disrupted sleep architecture