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In Laron syndrome patients, who have congenital IGF1 deficiency, the administration of IGF1 restores sexual maturation [17]

The present review summarizes the signaling mechanisms that contribute to drug abuse-related BBB dysfunction (Figure 1)

S-Acetyl-L-glutathione (50 M) increases intracellular GSH levels in primary fibroblasts derived from patients with glutathione synthetase deficiency

Through sacrificial oxidation, 7-DHC produces protective derivatives, such as 3,5-dihydroxycholest-7-en-6-one (DHCEO), which disrupt lipid peroxidation chain reactions and mitigate ferroptosis (Freitas et al., 2024