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By increasing TF activity, homocysteine would also enhance GPIIb-IIIa/fibrinogen-dependent platelet adhesion to endothelial cells (Dardik et al, 2000)

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This should also concern an in-depth analysis of the mechanism of action, including NO as the target most affected by the peptide, as well as the off-targets and their possible effects

Overexpression of the DJ-1 mutant associated with PD induces a significant increase in fragmented mitochondria, mitochondrial dysfunction, and increased neuronal vulnerability to oxidative stress [35]

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Cysteine, glycine, and glutamic acid combine to form the tripeptide GSH (13)