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However, PAI-1 carbonylation has been demonstrated to influence its functional activity and structural stability [38], therefore carbonylated PAI-1 may exhibit reduced activity, and we hypothesize that it could trigger a compensatory mechanism leading to an increase in its overall concentration observed in our cohort

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There is evidence that in APP overexpressing mice (TgCRND8 mice), there were increased levels of A at the synaptic terminal and extracellular accumulation of A, which were predominately located in the gray matter of the dorsal horn and the central part of dorsal column of the spinal cord