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J., Driscoll, K

Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimers disease models and human brains

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However, if the biological macromolecules have been denatured or experienced significant stress, the intended medicinal effects can become compromised

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While female mice were randomly cycling, we think it unlikely that SST-ergic-regulation of GHRH neuron firing closely follows the oestrous cycle, since the SST-ergic effect could be qualitatively different in GHRH neurons from a given mouse