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Sido et al, 1998), the increase in -GCS in mucosa from TNBS-treated rats can be interpreted as an adaptive response to control the consequences of the stress induced by TNBS as -GCS-HS is known to be upregulated in response to divergent stressful conditions including oxidative stress or GSH depletion (Morales et al, 1997, 1998

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the placebo was administered in the place of acetyl L-carnitine and/ or propionyl L-carnitine and/ or sildenafil

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Pericytes in diabetes-associated vascular disease

Hence, we evaluated the differentiation protocol efficiency by measuring the SIX2 + cells, a crucial MM NPC marker [55]