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Multiple studies have clearly shown that markers of oxidative stress such as malondialdehyde (MDA), superoxide dismutase (SOD) and lipid peroxidation byproduct levels are elevated in patients suffering from major depressive disorder (Sarandol et al., 2007

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Moreover, neutralizing HMGB1 with specific antibodies or depleting HMGB1 reduces inflammatory activation in macrophages, implying that HMGB1 inhibition may be a possible therapeutic strategy for controlling macrophage-mediated inflammation (103)

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We can indirectly assume ozone therapy influenced synthesis and release of cytokines (18,19) in such a way that a change from Th2 to Th1 (IL-2, IFNg) cytokine pattern occurs, or ozone therapy acted as Th2 cytokine inhibitor