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Based on available evidence, SCFAs exert protective effects by activating the Nrf2/GPX4 pathway to inhibit lipid peroxidation, while LPS acts through the dual mechanisms of suppressing the antioxidant defense system (GPX4) and disrupting iron metabolic homeostasis

Through transcriptional control of macrophage-expressed genes across diverse tissues and organs, ATFs are not only implicated in the initiation and progression of multiple disorders but also emerged as nodal regulators integrating pathophysiological signals with immune and metabolic reprogramming (Fig

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mRNA expression of inflammatory cytokine genes as measured by qRT-PCR