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Zhang H, Sun C, Sun Q, Li Y, Zhou C, Sun C

doi: 10.1136/gut.2007.129841

While challenging the importance of the protein binding hypothesis, it was instead suggested that the key mechanism of cell death involves P450-derived ROS formation during APAP metabolism, which triggers extensive LPO [59, 60]

Consequently, this implies that Letrozole possesses significant potential for inhibiting acetylcholinesterase and the therapeutic management of AD

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